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Hardell, Lennart
Publications (10 of 28) Show all publications
Carlberg, M., Koppel, T., Ahonen, M. & Hardell, L. (2017). Case-control study on occupational exposure to extremely low-frequency electromagnetic fields and glioma risk. American Journal of Industrial Medicine, 60(5), 494-503
Open this publication in new window or tab >>Case-control study on occupational exposure to extremely low-frequency electromagnetic fields and glioma risk
2017 (English)In: American Journal of Industrial Medicine, ISSN 0271-3586, E-ISSN 1097-0274, Vol. 60, no 5, p. 494-503Article in journal (Refereed) Published
Abstract [en]

BACKGROUND: Exposure to extremely low-frequency electromagnetic fields (ELF-EMF) was in 2002 classified as a possible human carcinogen, Group 2B, by the International Agency for Research on Cancer at WHO.

METHODS: Life time occupations were assessed in case-control studies during 1997-2003 and 2007-2009. An ELF-EMF Job-Exposure Matrix was used for associating occupations with ELF exposure (μT). Cumulative exposure (μT-years), average exposure (μT), and maximum exposed job (μT) were calculated.

RESULTS: Cumulative exposure gave for astrocytoma grade IV (glioblastoma multiforme) in the time window 1-14 years odds ratio (OR) = 1.9, 95% confidence interval (CI) = 1.4-2.6, p linear trend <0.001, and in the time window 15+ years OR = 0.9, 95%CI = 0.6-1.3, p linear trend = 0.44 in the highest exposure categories 2.75+ and 6.59+ μT years, respectively.

CONCLUSION: An increased risk in late stage (promotion/progression) of astrocytoma grade IV for occupational ELF-EMF exposure was found.

Place, publisher, year, edition, pages
John Wiley & Sons, 2017
Keywords
ELF-EMF, RF-EMF, astrocytoma, electromagnetic fields, glioma, occupational exposure, risk factors, wireless phones
National Category
Occupational Health and Environmental Health Cancer and Oncology
Identifiers
urn:nbn:se:oru:diva-57328 (URN)10.1002/ajim.22707 (DOI)000399609000010 ()28394434 (PubMedID)2-s2.0-85017463500 (Scopus ID)
Note

Funding agencies:

Kone Foundation Helsinki

Finland Cancer-och Allergifonden

Cancerhjälpen

Pandora-Foundation for Independent Research, Berlin, Germany

Available from: 2017-05-30 Created: 2017-05-30 Last updated: 2018-07-31Bibliographically approved
Hardell, L. (2017). Effects of Mobile Phones on Children's and Adolescents' Health: A Commentary. Child Development, 89(1), 137-140
Open this publication in new window or tab >>Effects of Mobile Phones on Children's and Adolescents' Health: A Commentary
2017 (English)In: Child Development, ISSN 0009-3920, E-ISSN 1467-8624, Vol. 89, no 1, p. 137-140Article in journal (Refereed) Published
Abstract [en]

The use of digital technology has grown rapidly during the last couple of decades. During use, mobile phones and cordless phones emit radiofrequency (RF) radiation. No previous generation has been exposed during childhood and adolescence to this kind of radiation. The brain is the main target organ for RF emissions from the handheld wireless phone. An evaluation of the scientific evidence on the brain tumor risk was made in May 2011 by the International Agency for Research on Cancer at World Health Organization. The scientific panel reached the conclusion that RF radiation from devices that emit nonionizing RF radiation in the frequency range 30 kHz-300 GHz is a Group 2B, that is, a "possible" human carcinogen. With respect to health implications of digital (wireless) technologies, it is of importance that neurological diseases, physiological addiction, cognition, sleep, and behavioral problems are considered in addition to cancer. Well-being needs to be carefully evaluated as an effect of changed behavior in children and adolescents through their interactions with modern digital technologies.

Place, publisher, year, edition, pages
John Wiley & Sons, 2017
National Category
Occupational Health and Environmental Health Human Computer Interaction Psychology (excluding Applied Psychology)
Identifiers
urn:nbn:se:oru:diva-60876 (URN)10.1111/cdev.12831 (DOI)000419895900014 ()28504422 (PubMedID)2-s2.0-85019250171 (Scopus ID)
Funder
Cancer and Allergy Foundation
Note

Funding Agency:

Pandora-Foundation for Independent Research, Berlin, Germany 

Available from: 2017-09-19 Created: 2017-09-19 Last updated: 2018-01-31Bibliographically approved
Carlberg, M. & Hardell, L. (2017). Evaluation of Mobile Phone and Cordless Phone Use and Glioma Risk Using the Bradford Hill Viewpoints from 1965 on Association or Causation. BioMed Research International, 2017, Article ID 9218486.
Open this publication in new window or tab >>Evaluation of Mobile Phone and Cordless Phone Use and Glioma Risk Using the Bradford Hill Viewpoints from 1965 on Association or Causation
2017 (English)In: BioMed Research International, ISSN 2314-6133, E-ISSN 2314-6141, Vol. 2017, article id 9218486Article in journal (Refereed) Published
Abstract [en]

Objective. Bradford Hill's viewpoints from 1965 on association or causation were used on glioma risk and use of mobile or cordless phones. Methods. All nine viewpoints were evaluated based on epidemiology and laboratory studies. Results. Strength: meta-analysis of case-control studies gave odds ratio (OR) = 1.90, 95% confidence interval (CI) = 1.31-2.76 with highest cumulative exposure. Consistency: the risk increased with latency, meta-analysis gave in the 10+ years' latency group OR = 1.62, 95% CI = 1.20-2.19. Specificity: increased risk for glioma was in the temporal lobe. Using meningioma cases as comparison group still increased the risk. Temporality: highest risk was in the 20+ years' latency group, OR = 2.01, 95% CI =1.41-2.88, for wireless phones. Biological gradient: cumulative use of wireless phones increased the risk. Plausibility: animal studies showed an increased incidence of glioma and malignant schwannoma in rats exposed to radiofrequency (RF) radiation. There is increased production of reactive oxygen species (ROS) from RF radiation. Coherence: there is a change in the natural history of glioma and increasing incidence. Experiment: antioxidants reduced ROS production from RF radiation. Analogy: there is an increased risk in subjects exposed to extremely low-frequency electromagnetic fields. Conclusion. RF radiation should be regarded as a human carcinogen causing glioma.

Place, publisher, year, edition, pages
Hindawi Publishing Corporation, 2017
National Category
Microbiology in the medical area Cancer and Oncology
Identifiers
urn:nbn:se:oru:diva-57327 (URN)10.1155/2017/9218486 (DOI)000398798300001 ()28401165 (PubMedID)2-s2.0-85016467564 (Scopus ID)
Note

Funding agencies:

Cancer-och Allergifonden  

Cancerhjalpen  

Pandora-Foundation for Independent Research, Berlin, Germany  

Available from: 2017-05-30 Created: 2017-05-30 Last updated: 2018-07-31Bibliographically approved
Hardell, L. (2017). World Health Organization, radiofrequency radiation and health: a hard nut to crack (Review). International journal of oncology, 51(2), 405-413
Open this publication in new window or tab >>World Health Organization, radiofrequency radiation and health: a hard nut to crack (Review)
2017 (English)In: International journal of oncology, ISSN 1791-2423, Vol. 51, no 2, p. 405-413Article in journal (Other academic) Published
Abstract [en]

In May 2011 the International Agency for Research on Cancer (IARC) evaluated cancer risks from radiofrequency (RF) radiation. Human epidemiological studies gave evidence of increased risk for glioma and acoustic neuroma. RF radiation was classified as Group 2B, a possible human carcinogen. Further epidemiological, animal and mechanistic studies have strengthened the association. In spite of this, in most countries little or nothing has been done to reduce exposure and educate people on health hazards from RF radiation. On the contrary ambient levels have increased. In 2014 the WHO launched a draft of a Monograph on RF fields and health for public comments. It turned out that five of the six members of the Core Group in charge of the draft are affiliated with International Commission on Non-Ionizing Radiation Protection (ICNIRP), an industry loyal NGO, and thus have a serious conflict of interest. Just as by ICNIRP, evaluation of non-thermal biological effects from RF radiation are dismissed as scientific evidence of adverse health effects in the Monograph. This has provoked many comments sent to the WHO. However, at a meeting on March 3, 2017 at the WHO Geneva office it was stated that the WHO has no intention to change the Core Group.

Place, publisher, year, edition, pages
Spandidos Publications, 2017
National Category
Cancer and Oncology
Identifiers
urn:nbn:se:oru:diva-60809 (URN)10.3892/ijo.2017.4046 (DOI)000405642300002 ()28656257 (PubMedID)2-s2.0-85021790903 (Scopus ID)
Funder
Cancer and Allergy Foundation
Note

Funding Agencies:

Cancerhjalpen 

Available from: 2017-10-10 Created: 2017-10-10 Last updated: 2018-08-06Bibliographically approved
Söderqvist, F., Carlberg, M. & Hardell, L. (2015). Biomarkers in volunteers exposed to mobile phone radiation. Toxicology Letters, 235(2), 140-146
Open this publication in new window or tab >>Biomarkers in volunteers exposed to mobile phone radiation
2015 (English)In: Toxicology Letters, ISSN 0378-4274, E-ISSN 1879-3169, Vol. 235, no 2, p. 140-146Article in journal (Refereed) Published
Abstract [en]

For some time it has been investigated whether low-intensity non-thermal microwave radiation from mobile phones adversely affects the mammalian blood-brain barrier (BBB). All such studies except one have been either in vitro or experimental animal studies. The one carried out on humans showed a statistically significant increase in serum transthyretin (TTR) 60 min after finishing of a 30-min microwave exposure session. The aim of the present study was to follow up on the finding of the previous one using a better study design. Using biomarkers analyzed in blood serum before and after the exposure this single blinded randomized counterbalanced study, including 24 healthy subjects aged 18-30 years that all underwent three exposure conditions (SAR(10G) = 2 W/kg, SAR(10G) = 0.2 W/kg, sham), tested whether microwaves from an 890-MHz phone-like signal give acute effects on the integrity of brain-shielding barriers. Over time, statistically significant variations were found for two of the three biomarkers (TTR; beta-trace protein); however, no such difference was found between the different exposure conditions nor was there any interaction between exposure condition and time of blood sampling. In conclusion this study failed to show any acute clinically or statistically significant effect of short term microwave exposure on the serum levels of S100 beta, TTR and b-trace protein with a follow up limited to two hours. The study was hampered by the fact that all study persons were regular wireless phone users and thus not naive as to microwave exposure. (C) 2015 Elsevier Ireland Ltd. All rights reserved.

Keywords
Blood-brain barrier; Cordless phones; Non-ionizing radiation; Radiofrequency fields; S100β, β-trace protein; Sleep; Tandomized trial; Transthyretin
National Category
Medical and Health Sciences
Research subject
Medicine
Identifiers
urn:nbn:se:oru:diva-44635 (URN)10.1016/j.toxlet.2015.03.016 (DOI)000353345500009 ()25839137 (PubMedID)2-s2.0-84926432189 (Scopus ID)
Note

Funding Agencies:

Cancer-hjälpen, Orebro University Hospital Cancer Fund, Cancer- och Allergifonden, Sweden

Instituto Nazionale Per L'Assicurazione Contro Gli Infortuni Sul Lavoro (I.N.A.I.L), Italy

Available from: 2015-05-18 Created: 2015-05-18 Last updated: 2017-12-04Bibliographically approved
Hardell, L. & Carlberg, M. (2015). Increasing rates of brain tumours in the swedish national inpatient register and the causes of death register. International Journal of Environmental Research and Public Health, 12(4), 3793-3813
Open this publication in new window or tab >>Increasing rates of brain tumours in the swedish national inpatient register and the causes of death register
2015 (English)In: International Journal of Environmental Research and Public Health, ISSN 1661-7827, E-ISSN 1660-4601, Vol. 12, no 4, p. 3793-3813Article in journal (Refereed) Published
Abstract [en]

Radiofrequency emissions in the frequency range 30 kHz-300 GHz were evaluated to be Group 2B, i.e., "possibly", carcinogenic to humans by the International Agency for Research on Cancer (IARC) at WHO in May 2011. The Swedish Cancer Register has not shown increasing incidence of brain tumours in recent years and has been used to dismiss epidemiological evidence on a risk. In this study we used the Swedish National Inpatient Register (IPR) and Causes of Death Register (CDR) to further study the incidence comparing with the Cancer Register data for the time period 1998-2013 using joinpoint regression analysis. In the IPR we found a joinpoint in 2007 with Annual Percentage Change (APC) +4.25%, 95% CI +1.98, +6.57% during 2007-2013 for tumours of unknown type in the brain or CNS. In the CDR joinpoint regression found one joinpoint in 2008 with APC during 2008-2013 +22.60%, 95% CI +9.68, +37.03%. These tumour diagnoses would be based on clinical examination, mainly CT and/or MRI, but without histopathology or cytology. No statistically significant increasing incidence was found in the Swedish Cancer Register during these years. We postulate that a large part of brain tumours of unknown type are never reported to the Cancer Register. Furthermore, the frequency of diagnosis based on autopsy has declined substantially due to a general decline of autopsies in Sweden adding further to missing cases. We conclude that the Swedish Cancer Register is not reliable to be used to dismiss results in epidemiological studies on the use of wireless phones and brain tumour risk.

Keywords
Brain tumour incidence; Cancer register; Causes of Death Register; Cordless phone; Mobile phone; Radiofrequency fields; Swedish National Inpatient Register
National Category
Environmental Sciences
Research subject
Enviromental Science
Identifiers
urn:nbn:se:oru:diva-44678 (URN)10.3390/ijerph120403793 (DOI)000353488500025 ()25854296 (PubMedID)2-s2.0-84927534730 (Scopus ID)
Funder
Cancer and Allergy Foundation
Note

Funding Agencies:

Cancerhjälpen

Pandora-Foundation for Independent Research, Berlin, Germany

Kone Foundation, Helsinki, Finland

Available from: 2015-05-22 Created: 2015-05-22 Last updated: 2018-06-30Bibliographically approved
Hardell, E., Kärrman, A., van Bavel, B., Bao, J., Carlberg, M. & Hardell, L. (2014). Case-control study on perfluorinated alkyl acids (PFAAs) and the risk of prostate cancer. Environment International, 63, 35-39
Open this publication in new window or tab >>Case-control study on perfluorinated alkyl acids (PFAAs) and the risk of prostate cancer
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2014 (English)In: Environment International, ISSN 0160-4120, E-ISSN 1873-6750, Vol. 63, p. 35-39Article in journal (Refereed) Published
Abstract [en]

Perfluorinated alkyl acids (PFAAs) are emerging environmental contaminants. Possible health effects for humans include increased risk for cancer but the knowledge is limited. In this study serum concentrations of certain perfluorinated sulfonates (PFHxS and PFOS) and carboxylates (PFOA, PFNA, PFDA, PFUnDA) were analyzed among 201 cases with prostate cancer and 186 population based control subjects. All blood samples were collected during 2007-2011 and no case had been treated with radio- or chemotherapy before enrolment in the study. The blood concentrations did not differ statistically significant between cases and controls except for PFDA with higher concentration among the cases (p = 0.03). Analyses based on Gleason score and prostate specific antigen (PSA) level did not change the results. Heredity was a risk factor for prostate cancer yielding odds ratio (OR) = 1.8, 95% confidence interval (CI) = 1.01-3.1. The analyzed PFAAs yielded statistically significant higher ORs in cases with a first degree relative reporting prostate cancer, e.g., PFOA gave OR = 2.6, 95% CI = 1.2-6.0 and PFOS gave OR = 2.7,95% CI = 1.04-6.8. The results showed a higher risk for prostate cancer in cases with heredity as a risk factor. In further studies interaction between gene and environment should be considered. (C) 2013 Elsevier Ltd. All rights reserved.

Place, publisher, year, edition, pages
Elsevier, 2014
Keywords
PFOS, PFOA, Heredity, Risk factors, Prostate cancer
National Category
Environmental Sciences
Research subject
Enviromental Science
Identifiers
urn:nbn:se:oru:diva-34263 (URN)10.1016/j.envint.2013.10.005 (DOI)000330909900005 ()24246240 (PubMedID)2-s2.0-84887579728 (Scopus ID)
Funder
Swedish Cancer Society
Note

Funding Agencies:

Cancerhjälpen

Odd Fellow Örebro

Örebro University Hospital Cancer Fund 

Available from: 2014-03-14 Created: 2014-03-13 Last updated: 2018-06-05Bibliographically approved
Carlberg, M. & Hardell, L. (2012). On the association between glioma, wireless phones, heredity and ionising radiation. Pathophysiology, 19(4), 243-252
Open this publication in new window or tab >>On the association between glioma, wireless phones, heredity and ionising radiation
2012 (English)In: Pathophysiology, ISSN 0928-4680, E-ISSN 1873-149X, Vol. 19, no 4, p. 243-252Article in journal (Refereed) Published
Abstract [en]

We performed two case–control studies on brain tumours diagnosed during 1 January 1997 to 30 June 2000 and 1 July 2000 to 31 December 2003, respectively. Living cases and controls aged 20–80 years were included. An additional study was performed on deceased cases with a malignant brain tumour using deceased controls. Pooled results for glioma yielded for ipsilateral use of mobile phone odds ratio (OR)=2.9, 95% confidence interval (CI)=1.8–4.7 in the >10 years latency group. The corresponding result for cordless phone was OR=3.8, 95% CI=1.8–8.1. OR increased statistically significant for cumulative use of wireless phones per 100h and per year of latency. For high-grade glioma ipsilateral use of mobile phone gave OR=3.9, 95% CI=2.3–6.6 and cordless phone OR=5.5, 95% CI=2.3–13 in the >10 years latency group. Heredity for brain tumour gave OR=3.4, 95% CI=2.1–5.5 for glioma. There was no interaction with use of wireless phones. X-ray investigation of the head gave overall OR=1.3, 95% CI=1.1–1.7 for glioma without interaction with use of wireless phones or heredity. In conclusion use of mobile and cordless phone increased the risk for glioma with highest OR for ipsilateral use, latency >10 years and third tertile of cumulative use in hours. In total, the risk was highest in the age group <20 years for first use of a wireless phone.

Place, publisher, year, edition, pages
Elsevier, 2012
Keywords
Brain tumours, Glioma, Mobile phone, Cordless phone, Risk factors, Interaction
National Category
Cancer and Oncology
Identifiers
urn:nbn:se:oru:diva-58381 (URN)10.1016/j.pathophys.2012.07.001 (DOI)2-s2.0-84868666111 (Scopus ID)
Funder
Swedish Cancer Society
Note

Funding Agencies:

Allergifonden

Cancerhjälpen

Örebro University Hospital Cancer Fund

Available from: 2017-06-29 Created: 2017-06-29 Last updated: 2017-10-18Bibliographically approved
Behrens, T., Lynge, E., Cree, I., Lutz, J.-M., Eriksson, M., Guenel, P., . . . Ahrens, W. (2012). Pesticide exposure in farming and forestry and the risk of uveal melanoma. Cancer Causes and Control, 23(1), 141-151
Open this publication in new window or tab >>Pesticide exposure in farming and forestry and the risk of uveal melanoma
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2012 (English)In: Cancer Causes and Control, ISSN 0957-5243, E-ISSN 1573-7225, Vol. 23, no 1, p. 141-151Article in journal (Refereed) Published
Abstract [en]

Since pesticides are disputed risk factors for uveal melanoma, we studied the association between occupational pesticide exposure and uveal melanoma risk in a case-control study from nine European countries.

Incident cases of uveal melanoma and population as well as hospital controls were included and frequency-matched by country, 5-year age groups and sex. Self-reported exposure was quantified with respect to duration of exposure and pesticide application method. We calculated the exposure intensity level based on application method and use of personal protective equipment. Odds ratios (OR) and 95% confidence intervals (95% CI) were estimated by unconditional logistic regression analyses and adjusted for several potential confounders.

293 case and 3,198 control subjects were interviewed. We did not identify positive associations with activities in farming or forestry, pesticide application or pesticide mixing. No consistent positive associations were seen with exposure intensity level scores either. The only statistically significantly raised association in this study was for exposure to chemical fertilizers in forestry (OR = 8.93; 95% CI 1.73-42.13), but this observation was based on only six exposed subjects. Results did not change when we restricted analyses to morphologically verified cases and excluded proxy interviews as well as cancer controls. We did not observe effect modification by sex or eye color.

Risk estimates for pesticide exposures and occupational activities in agriculture and forestry were not increased and did not indicate a hormonal mechanism due to these exposures.

Place, publisher, year, edition, pages
Springer, 2012
Keywords
Agriculture, Animal husbandry, Chemical fertilizers, Multicenter study, Ocular melanoma
National Category
Agricultural Occupational Health and Safety Cancer and Oncology
Identifiers
urn:nbn:se:oru:diva-58344 (URN)10.1007/s10552-011-9863-z (DOI)000297757400014 ()22052342 (PubMedID)2-s2.0-82955195606 (Scopus ID)
Funder
Swedish Society of Medicine
Note

Funding Agencies:

European Commission  

DGXII  

Program BIOMED  BMH1 CT 93-1630  ERB CIPD CT 940285 

The strategic Environment Program  92.01.015.7-06 

Danish Epidemiology Science Center  

Danish National Research Foundation  

Ligue Nationale contre le cancer  

Institut National de la Sante et de la Recherche Medicale (INSERM)  4R006A 

French Ministry of Environment  237.01. 94.40182 

Federal Ministry for Education, Science, Research and Technology (BMBF)  01-HP-684/8 

The Italian Association for Cancer Research (AIRC)  

Special Project Oncology  

Compagnia di San Paolo/FIRMS  

MURST, Region Piedmont  

Fondo de Investigacion de la Sanitarie  

Ministerio de Sanidad y Consumo  

Unidad de Investigacion Clinico-Epidemiologica  

Hospital Dr. Peset. Generalitat Valenciana  FISS. 95/0044-01, 96/0043-01 

Departmento de Sanidad y Consumo, Gobierno Vasco  

Fondo de Investigacion de la Sanitaria  

Ayuda a la Investigacion del Departamento de Salud del Gobierno de Navarra  

Swedish Council for Work Life Research  

Research Foundation of the Department of Oncology in Umea  

Lund University Hospital Research Foundation, Gunnar, Arvid  

Elisabeth Nilsson Cancer Foundation  

Örebro County Council Research Committee  

Örebro Medical Center Research Foundation  

John and Augusta Persson Foundation for Scientific Medical Research  

Berta Kamprad Foundation for Cancer Research  

Federation Nationale des Centres de Lutte contre le Cancer  

Fondation de France  955368 

Available from: 2017-06-28 Created: 2017-06-28 Last updated: 2018-05-14Bibliographically approved
Hardell, K., Carlberg, M., Hardell, L., Björnfoth, H., Ericson Jogsten, I., Eriksson, M., . . . Lindström, G. (2009). Concentrations of organohalogen compounds and titres of antibodies to Epstein-Barr virus antigens and the risk for non-Hodgkin lymphoma. Oncology Reports, 21(6), 1567-1576
Open this publication in new window or tab >>Concentrations of organohalogen compounds and titres of antibodies to Epstein-Barr virus antigens and the risk for non-Hodgkin lymphoma
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2009 (English)In: Oncology Reports, ISSN 1021-335X, E-ISSN 1791-2431, Vol. 21, no 6, p. 1567-1576Article in journal (Refereed) Published
Abstract [en]

Exposure to some pesticides and persistent organic pollutants (POPs) has been indicated to be a risk factor for non-Hodgkin's lymphoma (NHL). Epstein-Barr virus (EBV) has been associated with some subgroups of NHL. In a previous study we found an interaction between high concentrations of some POPs and titres of antibodies to EBV early antigen (EA IgG) in relation to NHL. In the present study we measured lipid adjusted plasma concentrations of 35 congeners of polychlorinated biphenyls (PCB), p,p'- dichlorodiphenyldichloroethyelene (p,p'-DDE), hexachlorobenzene (HCB), seven subgroups of chlordanes (cisheptachlorepoxide, cis-chlordane, trans-chlordane, oxychlordane, MC6, trans-nonachlordane, cis-nonachlordane) and one polybrominated diphenylether (PBDE) congener (no. 47) in 99 cases with NHL and 99 population based controls. Odds ratios (OR) for NHL were estimated. Sum of PCBs > median in the controls gave odds ratio (OR) 2.0, 95% confidence interval (CI) 0.99-3.9. High sum of chlordanes yielded OR 2.3, 95% CI 1.2-4.5. An interaction with EBV EA IgG was found. High sum of PCB gave OR 5.2, 95% CI 1.9-14 in the group with EA IgG > 40. Similarly HCB yielded OR 5.3, 95% CI 1.9-15, pp'-DDE gave OR 3.3, 95% CI 1.4-7.7 and sum of chlordanes yielded OR 6.8, 95% CI 2.3-20, whereas no association was found with PBDE. In summary, thisstudy confirmed an association between certain POPs andNHL with an interaction with titre of IgG antibody to EBV EA.

Place, publisher, year, edition, pages
Athens, Greece: Spandidos Publications, 2009
Keywords
Non-Hodgkin lymphoma, polychlorinated biphenyls, persistent organic pollutants, Epstein-Barr virus, aetiology
National Category
Chemical Sciences
Identifiers
urn:nbn:se:oru:diva-47989 (URN)10.3892/or_00000389 (DOI)000266185900029 ()19424638 (PubMedID)2-s2.0-67649987698 (Scopus ID)
Available from: 2016-02-05 Created: 2016-02-05 Last updated: 2017-11-30Bibliographically approved
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