oru.sePublikasjoner
Endre søk
RefereraExporteraLink to record
Permanent link

Direct link
Referera
Referensformat
  • apa
  • harvard1
  • ieee
  • modern-language-association-8th-edition
  • vancouver
  • Annet format
Fler format
Språk
  • de-DE
  • en-GB
  • en-US
  • fi-FI
  • nn-NO
  • nn-NB
  • sv-SE
  • Annet språk
Fler språk
Utmatningsformat
  • html
  • text
  • asciidoc
  • rtf
Lack of neurohumoral response to pneumoperitoneum for laparoscopic cholecystectomy
Dept. Anesthesia and Intensive Care, Huddinge University Hospital, Huddinge, Sweden.
Örebro universitet, Institutionen för medicinska vetenskaper. Region Örebro län. Department of Surgery, Huddinge University Hospital, Huddinge, Sweden.ORCID-id: 0000-0003-2636-4745
Department of Surgery, Karolinska Hospital,6 Stockholm, Sweden.
Dept. Anesthesia and Intensive Care, Huddinge University Hospital, Huddinge, Sweden.
1998 (engelsk)Inngår i: Surgical Endoscopy, ISSN 0930-2794, E-ISSN 1432-2218, Vol. 12, nr 10, s. 1217-1223Artikkel i tidsskrift (Fagfellevurdert) Published
Abstract [en]

Background: Pneumoperitoneum (PP) for laparoscopic surgery induces prompt changes in circulatory parameters. The rapid onset of these changes suggests a reflex origin, and the present study was undertaken to evaluate whether release of vasopressor substances could be responsible for these alterations. The influence of two different anesthesia techniques was also evaluated. Methods: American Society of Anesthesiologists (ASA) class I patients, scheduled for laparoscopic cholecystectomy, were investigated. The first group (n = 10) was anesthetized intravenously. The second group (n = 6) had inhalation anesthesia. Plasma vasopressin, catecholamines, and plasma renin activity were investigated as neurohumoral vasopressor markers of circulatory stress. The general stress response to surgery was assessed by analysis of plasma cortisol. Results: Induction of pneumoperitoneum caused no apparent activation of vasopressor substances, although several hemodynamic parameters responded promptly. Conclusion: The hemodynamic alterations, seen at the establishment of PP during stable anesthesia, cannot be explained by elevation of vasopressor substances in circulating blood.

sted, utgiver, år, opplag, sider
Springer-Verlag New York, 1998. Vol. 12, nr 10, s. 1217-1223
HSV kategori
Identifikatorer
URN: urn:nbn:se:oru:diva-63888DOI: 10.1007/s004649900824ISI: 000076104900006PubMedID: 9745060Scopus ID: 2-s2.0-0032185983OAI: oai:DiVA.org:oru-63888DiVA, id: diva2:1171085
Tilgjengelig fra: 2018-01-05 Laget: 2018-01-05 Sist oppdatert: 2018-02-05bibliografisk kontrollert

Open Access i DiVA

Fulltekst mangler i DiVA

Andre lenker

Forlagets fulltekstPubMedScopus

Personposter BETA

Ljungqvist, Olle

Søk i DiVA

Av forfatter/redaktør
Ljungqvist, Olle
Av organisasjonen
I samme tidsskrift
Surgical Endoscopy

Søk utenfor DiVA

GoogleGoogle Scholar

doi
pubmed
urn-nbn

Altmetric

doi
pubmed
urn-nbn
Totalt: 127 treff
RefereraExporteraLink to record
Permanent link

Direct link
Referera
Referensformat
  • apa
  • harvard1
  • ieee
  • modern-language-association-8th-edition
  • vancouver
  • Annet format
Fler format
Språk
  • de-DE
  • en-GB
  • en-US
  • fi-FI
  • nn-NO
  • nn-NB
  • sv-SE
  • Annet språk
Fler språk
Utmatningsformat
  • html
  • text
  • asciidoc
  • rtf