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Hyperosmotic glucose infusion during hemorrhage does not reduce bacterial translocation in 24 hour-starved rats
Department of Surgery, Karolinska Hospital, Stockholm, Sweden; Microbiology and Tumorbiology Center, Karolinska Institute, Stockholm, Sweden.
Department of Surgery, Karolinska Hospital, Stockholm, Sweden; Microbiology and Tumorbiology Center, Karolinska Institute, Stockholm, Sweden.
Department of Surgery, Karolinska Hospital, Stockholm, Sweden; Microbiology and Tumorbiology Center, Karolinska Institute, Stockholm, Sweden.
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1995 (English)In: Shock, ISSN 1073-2322, E-ISSN 1540-0514, Vol. 4, no 2, p. 113-116Article in journal (Refereed) Published
Abstract [en]

Food deprivation 24 h before stress increases bacterial translocation in hemorrhage. Presently it tested whether hyperosmolality, induced by exogenous glucose infusion to improve plasma refill, prevents or reduces bacterial translocation after experimental hemorrhage in 24 h food-deprived rats. Rats were given an i.v. infusion of either 2 mL of 30% glucose (G) or the same volume of .9% NaCl (C) while simultaneously being submitted to a standardized 60 min hemorrhage period, of moderate or more severe hemorrhage. Blood was not reinfused. Despite development of marked hyperglycemia (p < .001, G vs. C) resulting in significantly greater reductions in packed cell volume (p < .001, G vs. C), bacterial translocation was detected similarly in both groups regardless of whether moderate (10/12-G, 9/12-C) or severe (15/19-G, 15/18-C) hemorrhage was inflicted. It was concluded that hyperglycemic hyperosmolality did not prevent bacterial translocation in these models of hemorrhagic stress in 24 h-starved rats.

Place, publisher, year, edition, pages
Lippincott Williams & Wilkins, 1995. Vol. 4, no 2, p. 113-116
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Medical and Health Sciences Physiology
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URN: urn:nbn:se:oru:diva-63857DOI: 10.1097/00024382-199508000-00006ISI: A1995RN50000007PubMedID: 7496895Scopus ID: 2-s2.0-0029348014OAI: oai:DiVA.org:oru-63857DiVA, id: diva2:1170974
Available from: 2018-01-05 Created: 2018-01-05 Last updated: 2018-02-05Bibliographically approved

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Ljungqvist, Olle

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