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Longitudinal cerebrospinal fluid biomarker trajectories along the Alzheimer's disease continuum in the BIOMARKAPD study
Hospital de la Santa Creu i Sant Pau, Biomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain; Centre of Biomedical Investigation Network for Neurodegenerative Diseases (CIBERNED), Madrid, Spain.
Hospital de la Santa Creu i Sant Pau, Biomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain; Centre of Biomedical Investigation Network for Neurodegenerative Diseases (CIBERNED), Madrid, Spain.
Center for Research and Advanced Therapies, Fundación CITA-alzheimer Fundazioa, San Sebastian, Spain.
Amsterdam UMC, Department of Neurology and Alzheimer Center, VU University Medical Center, Neuroscience Campus Amsterdam, Amsterdam, the Netherlands.
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2019 (English)In: Alzheimer's & Dementia: Journal of the Alzheimer's Association, ISSN 1552-5260, E-ISSN 1552-5279, Vol. 15, no 6, p. 742-753Article in journal (Refereed) Published
Abstract [en]

INTRODUCTION: Within-person trajectories of cerebrospinal fluid (CSF) biomarkers in Alzheimer's disease (AD) are not well defined.

METHODS: We included 467 subjects from the BIOMARKAPD study with at least two serial CSF samples. Diagnoses were subjective cognitive decline (n = 75), mild cognitive impairment (n = 128), and AD dementia (n = 110), and a group of cognitively unimpaired subjects (n = 154) were also included. We measured baseline and follow-up CSF levels of total tau (t-tau), phosphorylated tau (p-tau), YKL-40, and neurofilament light (NfL). Median CSF sampling interval was 2.1 years.

RESULTS: CSF levels of t-tau, p-tau, NfL, and YKL-40 were 2% higher per each year of baseline age in controls (P <.001). In AD, t-tau levels were 1% lower (P <.001) and p-tau levels did not change per each year of baseline age. Longitudinally, only NfL (P <.001) and YKL-40 (P <.02) increased during the study period.

DISCUSSION: All four CSF biomarkers increase with age, but this effect deviates in AD for t-tau and p-tau.

Place, publisher, year, edition, pages
Elsevier, 2019. Vol. 15, no 6, p. 742-753
Keywords [en]
Alzheimer, Amyloid, CSF, Inflammation, Neurofilaments, Tau, YKL-40
National Category
Medical and Health Sciences Neurology
Identifiers
URN: urn:nbn:se:oru:diva-79152DOI: 10.1016/j.jalz.2019.01.015ISI: 000470084500002PubMedID: 30967340Scopus ID: 2-s2.0-85063904370OAI: oai:DiVA.org:oru-79152DiVA, id: diva2:1385393
Funder
Swedish Research CouncilEU, European Research Council, 681712
Note

Funding Agencies:

Ministry of Health, Italy, Appeared in article as Italian Ministry of Health

Netherlands Organization for Health Research and Development, Appeared in article as Netherlands Organization for Health, Research and Development (ZonMw)

Instituto de Salud Carlos III, Appeared in article as Instituto de Salud Carlos III, Grant numbers PI14/1561, PI17/1895

Fondo Europeo de Desarrollo Regional (FEDER), Union Europea, "Una manera de hacer Europa"

Federal Ministry of Education & Research (BMBF), Appeared in article as Bundesministerium fur Bildung und Forschung (BMBF), Germany

Swedish Research Council, Appeared in article as Swedish Research Council

Innovation Fund Denmark, Grant number 0603-00470B

Fonds de Recherche du Quebec-Sante (FRQS)

J. L. Levesque Foundation  

Department of Health of the Basque Government, Grant number 2016111096

Instituto de Salud Carlos III, Appeared in article as Carlos III Institute of Health, Grant number PI15/00919

"Obra Social Kutxa Fundazioa"

European Research Council (ERC), Appeared in article as European Research Council, Grant number 681712

Swedish Research Council, Appeared in article as Swedish Research Council, Grant number 2013-2546

Torsten Soderberg Foundation at the Royal Swedish Academy of Sciences  

European Union (EU), Appeared in article as European Commission as part of the 6th Framework Program, Grant number 37670

Available from: 2020-01-14 Created: 2020-01-14 Last updated: 2023-03-28Bibliographically approved

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Freund-Levi, Yvonne

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