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  • 1.
    Ludvigsson, Jonas F.
    et al.
    Örebro University, School of Medical Sciences. Örebro University Hospital. Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm; Department of Paediatrics, Örebro University Hospital, Örebro; Division of Epidemiology and Public Health, School of Medicine, University of Nottingham, City Hospital, Nottingham, UK; Department of Medicine, Columbia University College of Physicians and Surgeons, New York, NY.
    Jarrick, Simon
    Örebro University, School of Medical Sciences. Department of Paediatrics.
    Murray, Joseph A.
    Department of Immunology, Division of Gastroenterology and Hepatology, Mayo Clinic, Rochester MN, USA.
    Emilsson, Louise
    Department of Health Management and Health Economy, Institute of Health and Society, University of Oslo, Oslo, Norway; Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston MA, USA; Primary Care Research Unit, Vårdcentralen Värmlands Nysäter, Värmland County, Sweden.
    Celiac Disease and Risk of Henoch-Schonlein Purpura Population-based Cohort Study2018In: Journal of Clinical Gastroenterology, ISSN 0192-0790, E-ISSN 1539-2031, Vol. 52, no 2, p. 141-145Article in journal (Refereed)
    Abstract [en]

    Background and Aims: A recent study found a 10-fold increased risk of celiac disease (CD) in individuals with Henoch-Schonlein purpura (HSP), but the confidence interval (CI) was wide.

    Methods: The retrospective cohort study of all patients with CD in Sweden, diagnosed through small intestinal biopsy from 1969 to 2008 (n = 29,077). Each individual with CD was matched to up to 5 controls (n = 144,433). Data on study participants were linked to diagnostic codes for HSP in the National Patient Registry. Through Cox regression we estimated hazard ratios for CD and later HSP. Through logistic regression we calculated odds ratios for HSP preceding CD.

    Results: During follow-up 19 individuals with CD and 99 controls developed HSP. This corresponded to a hazard ratio of 0.96 (95% CI, 0.59-1.56). Looking backward, we found no increased risk of earlier HSP in patients with CD (odds ratio = 1.02; 95% CI, 0.601.72).

    Conclusions: In this study of more than 29,000 patients with CD, we found no increased risk of HSP before or after CD.

  • 2.
    Welander, Adina
    et al.
    Dept Med, Clin Epidemiol Unit, Karolinska Univ Hosp, Stockholm, Sweden.
    Sundelin, Birgitta
    Dept Pathol, Karolinska Univ Hosp, Stockholm, Sweden.
    Fored, Michael
    Dept Med, Clin Epidemiol Unit, Karolinska Univ Hosp, Stockholm, Sweden.
    Ludvigsson, Jonas F.
    Örebro University Hospital. Dept Pediat.
    Increased Risk of IgA Nephropathy Among Individuals With Celiac Disease2013In: Journal of Clinical Gastroenterology, ISSN 0192-0790, E-ISSN 1539-2031, Vol. 47, no 8, p. 678-683Article in journal (Refereed)
    Abstract [en]

    Goal: To determine the risk of future biopsy-verified IgA nephropathy (IgAN) among individuals with biopsy-verified celiac disease (CD). Background: Individuals with CD suffer increased risk of end-stage renal disease. An association between CD and IgAN has been suggested; however, results have been inconclusive and no previous study has considered the risk of IgAN in biopsy-verified CD. Study: We performed a population-based prospective cohort study. We identified 27,160 individuals with CD (Marsh stage III) and no previous renal disease through small-intestinal biopsy reports obtained between July 1969 and February 2008 in all (n=28) Swedish pathology departments. Individuals with IgAN were identified by biopsy reports acquired at the 4 Swedish pathology departments specialized in renal pathology. Cox regression analysis was used to determine the risk of future IgAN among individuals with CD compared with 133,949 age-matched and sex-matched reference individuals. Results: Seven (0.026%) individuals with CD and 11 (0.008%) reference individuals developed IgAN. We found an increased risk of biopsy-verified IgAN among individuals with CD [hazard ratio, 3.03; 95% confidence interval, 1.22-7.56]. The risk increase remained statistically significant after adjustment for prior liver disease and country of birth. Conclusions: Individuals with CD suffer a 3-fold increased risk of future IgAN. Our findings warrant awareness of renal function in individuals with CD.

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