To Örebro University

Incarceration is a disruptive event that is experienced by a considerable proportion of the United States population. Research has identified social factors that predict incarceration risk, but scholars have called for a focus on the ways that individual differences combine with social factors to affect incarceration risk. Our study is an initial attempt to heed this call using whole-genome data. We use data from the Health and Retirement Study (HRS) (N = 6716) to construct a genome-wide measure of genetic propensity for aggressive behavior and use it to predict lifetime incarceration risk. We find that participants with a higher genetic propensity for aggression are more likely to experience incarceration, but the effect is stronger for males than females. Importantly, we identify a gene-environment interaction (G x E)-genetic propensity is reduced, substantively and statistically, to a non-significant predictor for males raised in homes where at least one parent graduated high school. We close by placing these findings in the broader context of concerns that have been raised about genetics research in criminology.

Past Swedish research has shown that immigrants arriving in the receiving country at an older age are less likely to commit crime than immigrants arriving at a younger age. Segmented assimilation theory argues that the family and neighborhood may be important factors affecting how age at immigration and crime are related to one another. This study used population-based register data on foreign-background males from Stockholm to test the effect of age at immigration on crime. Potential confounding from. the family and neighborhood was addressed using variables and modeling strategies. Initial results, using variables to control for confounding, showed that people who immigrated around age 4 were the most likely to be suspected of a crime. When controlling for unmeasured family characteristics, it seemed that a later age at immigration was tied to a lower likelihood of crime, which does not corroborate past research findings. The effect of age at immigration, however, was not statistically significant. The results imply that future research on entire families may be a worthwhile endeavor.

This study uses Swedish register data to assess the impact of war in the home country on the individual likelihood of registered violent crime among young male immigrants in Stockholm, Sweden. War in the home country during a migrant’s residence is significantly related to a higher likelihood of registration for a violent crime. However, these results were not sustained in a sensitivity analysis, which considered serious property crime. Analysis of the history of war in the home country produces effects opposite to those predicted, with more years of war reducing the likelihood of violent crime. These findings indicate that war is capturing other factors, within the home or the receiving country, that may be related to violent crime.

Immigration policies that attach citizenship and deportation consequences to crime may be aimed at deterring crime, but they also effectively marginalize immigrants and may promote crime. Evidence from Sweden and around the world indicates that, where citizenship is concerned, marginalization may have won out. This research used a population-based sample of approximately 20,000 Swedish males and more rigorous methods than past studies to test the effects of citizenship and region of origin on official police suspicion for a serious crime. The findings showed that a lack of citizenship is related to greater involvement in crime, indicating support for the marginalizing effects of immigration policies. Yet, the region of origin results presented a conflicting picture in which neither ideas on deterrence nor marginalization could be supported. In conclusion, neither the potential deterrent effects of immigration policy nor its marginalizing effects were strongly supported.

Vilken roll spelar bostadsområden för graden av brottslighet? Amber Beckley, lektor i kriminologi vid Stockholms universitet, och hennes team ville sätta tidigare forskning om utländsk bakgrund och brottslighet i ett nytt ljus. Resultaten överraskade. Studien ingår i "Det svenska tillståndet - en antologi om brottsutvecklingen i Sverige". Inspelat den 5 maj 2022 på Piperska muren i Stockholm. Arrangör: Institutet för framtidsstudier.

This doctoral thesis considers how factors from the home country, the family, and the individual impact the risk for criminal offending among young males from a foreign background residing in Stockholm. I use Swedish register data to examine the risk for police registered suspicion of criminal offending. The introductory chapter presents an historical overview of immigration in Sweden, theories of criminal offending, and details about analysis of register data. It is followed by three empirical studies that consider unique risk factors for crime among children of immigrants while controlling for factors encountered within Sweden. The first study shows that young male children of immigrants do not seem to be inherently violent as a result of coming from a war-torn country. The second study indicates that it is not the age at immigration, but the family situation that seems to dictate criminal propensity. The final study suggests that threats of deportation and stricter immigration policies do not seem to deter criminality. The most interesting result was probably that high home country human development was a protective factor against crime. This is the first known work to uncover such a result. Future theoretical development may be best aimed at unpacking and empirically evaluating the human development index as a risk factor. Together, these three studies suggest that some previously unconsidered uniquely immigrant factors are related to risk for criminality. 

Black men, especially those in the lower-class are stereotyped as hypermasculine. Such stereotypes may affect placement in criminal justice programs as well as whether offenders successfully complete programming given placement. This article considers whether the intersection of class and race affect boot camp failure. Using data from MacKenzie's evaluation of correctional boot camps, results show that neither race, nor the interaction of race with indicators of orientation towards decent or street behavior has any effect on dropping out of boot camp. What is significant in predicting boot camp completion is the offender's belief prior to beginning the program that he is "tough enough" to handle the institution. However, this result is opposite of expected for black men. The implications of this result and future directions are considered. 

Purpose: It is well-established that victims and offenders are often the same people, a phenomenon known as the victim-offender overlap, but the developmental nature of this overlap remains uncertain. In this study, we drew from a developmental theoretical framework to test effects of genetics, individual characteristics, and routine-activity-based risks. Drawing from developmental literature, we additionally tested the effect of an accumulation of adverse childhood experiences (ACEs).

Methods: Data came from the Environmental Risk (E-Risk) Study, a representative UK birth cohort of 2232 twins born in 1994-1995 and followed to age 18 (with 93% retention). Crime victimization and offending were assessed through self-reports at age 18 (but findings replicated using crime records). We used the classical twin study method to decompose variance in the victim-offender overlap into genetic and environmental components. We used logistic regression to test the effects of childhood risk factors.

Results: In contrast to past twin studies, we found that environment (as well as genes) contributed to the victim-offender overlap. Our logistic regression results showed that childhood low self-control and childhood antisocial behavior nearly doubled the odds of becoming a victim-offender, compared to a victim-only or an offender-only. Each additional ACE increased the odds of becoming a victim-offender, compared to a victim-only or an offender-only, by approximately 12%, pointing to the importance of cumulative childhood adversity.

Conclusions: This study showed that the victim-offender overlap is, at least partially, developmental in nature and predictable from personal childhood characteristics and an accumulation of many adverse childhood experiences.

Importance:  Lead is a neurotoxin with well-documented effects on health. Research suggests that lead may be associated with criminal behavior. This association is difficult to disentangle from low socioeconomic status, a factor in both lead exposure and criminal offending.

Objective:  To test the hypothesis that a higher childhood blood lead level (BLL) is associated with greater risk of criminal conviction, recidivism (repeat conviction), conviction for violent offenses, and variety of self-reported criminal offending in a setting where BLL was not associated with low socioeconomic status.

Design, Setting, and Participants:  A total of 553 individuals participated in a prospective study based on a population-representative cohort born between April 1, 1972, and March 31, 1973, from New Zealand; the Dunedin Multidisciplinary Health and Development Study observed participants to age 38 years (December 2012). Statistical analysis was performed from November 10, 2016, to September 5, 2017.

Exposures:  Blood lead level measured at age 11 years.

Main Outcomes and Measures:  Official criminal conviction cumulative to age 38 years (data collected in 2013), single conviction or recidivism, conviction for nonviolent or violent crime, and self-reported variety of crime types at ages 15, 18, 21, 26, 32, and 38 years.

Results: Participants included 553 individuals (255 female and 298 male participants) who had their blood tested for lead at age 11 years. The mean (SD) BLL at age 11 years was 11.01 (4.62) μg/dL. A total of 154 participants (27.8%) had a criminal conviction, 86 (15.6%) had recidivated, and 53 (9.6%) had a violent offense conviction. Variety scores for self-reported offending ranged from 0 to 10 offense types at each assessment; higher numbers indicated greater crime involvement. Self-reported offending followed the well-established age-crime curve (ie, the mean [SD] variety of self-reported offending increased from 1.99 [2.82] at age 15 years to its peak of 4.24 [3.15] at age 18 years and 4.22 [3.02] at age 21 years and declined thereafter to 1.10 [1.59] at age 38 years). Blood lead level was a poor discriminator between no conviction and conviction (area under the curve, 0.58). Overall, associations between BLL and conviction outcomes were weak. The estimated effect of BLL was lower for recidivism than for single convictions and lower for violent offending than for nonviolent offending. Sex-adjusted associations between BLL reached statistical significance for only 1 of the 6 self-reported offending outcomes at age 15 years (r = 0.10; 95% CI, 0.01-0.18; P = .02).

Conclusions and Relevance:  This study overcomes past limitations of studies of BLL and crime by studying the association in a place and time where the correlation was not confounded by childhood socioeconomic status. Findings failed to support a dose-response association between BLL and consequential criminal offending.

In this study we considered the effect of childhood risk factors on adolescent victimization, measured at 18 years of age. We analyzed 1000 twipairs born 1994–1996 who were members of the Environmental Risk (E-Risk) Longitudinal Study, a prospective cohort study from the United Kingdom. We considered risk factors from two developmental periods (early childhood and early adolescence) and across multiple domains (individual, family, neighborhood): IQ, under controlled temperament, parent antisocial behavior, severe childhood victimization from ages 5 to 12, neighborhood socioeconomic, and neighborhood social cohesion. These childhood and early adolescence risk factors were tied to adolescent victimization. Many of these risk factors, however, are known risk factors for criminal behavior. Additionally, criminal behavior and victimization have been shown to be correlated in past research and were correlated the present sample of 18 year-olds (r = .40). We thus reanalyzed the relationship between the risk factors and victimization while controlling for adolescent offending. We found that the effect size for many of the childhood risk factors for victimization decreased and some became non-significant, suggesting that the risk factors described adolescents who were both victims and perpetrators. However, we found that one of the most robust predictors of adolescent victimization was childhood victimization, implying a continuity in victimization risk over the life-course. This presentation will include a comparison for twins discordant for severe childhood victimization.

Purpose: To describe official adult-onset offenders, investigate their antisocial histories and test hypotheses about their origins.

Methods: We defined adult-onset offenders among 931 Dunedin Study members followed to age 38, using criminal-court conviction records.

Results: Official adult-onset offenders were 14% of men, and 32% of convicted men, but accounted for only 15% of convictions. As anticipated by developmental theories emphasizing early-life influences on crime, adult-onset offenders' histories of antisocial behavior spanned back to childhood. Relative to juvenile-offenders, during adolescence they had fewer delinquent peers and were more socially inhibited, which may have protected them from conviction. As anticipated by theories emphasizing the importance of situational influences on offending, adult onset offenders, relative to non-offenders, during adulthood more often had schizophrenia, bipolar disorder, and alcohol-dependence, had weaker social bonds, anticipated fewer informal sanctions, and self-reported more offenses. Contrary to some expectations, adult-onset offenders did not have high IQ or high socioeconomic-status families protecting them from juvenile conviction.

Conclusions: A tailored theory for adult-onset offenders is unwarranted because few people begin crime de novo as adults. Official adult-onset offenders fall on a continuum of crime and its correlates, between official non offenders and official juvenile-onset offenders. Existing theories can accommodate adult-onset offenders.

This chapter discusses immigration and criminality in Sweden. It presents a broad overview of immigration, immigrants and crime in Europe in general, which serves as an important point of departure for comparing and contrasting the Swedish context. The chapter covers the history of immigration to Sweden along with Sweden’s development of immigration and integration policy, and discusses Sweden’s immigration and integration policy. It provides context on studies of immigrant crime in Sweden. The section that follows provides an overview on immigrant criminality in Sweden based on research that has been done since the 1970s. Finally, the chapter summarizes and offer conclusions on the outlook of immigrant criminality in Sweden. Overall, the research indicates that over-representation in recorded crime has remained stable over time, even if immigration to Sweden has changed in character. The immigrant criminality in Sweden appears to depend mostly on Sweden’s reaction to and reception of immigrants.

Much is known about the patterning of offending throughout life, but less about the patterning of victimization. In this study, we used data from the Stockholm Life-Course Project (SLCP), a longitudinal study that includes measures of childhood problem behaviour. We analysed offending (criminal conviction and police suspicion), inpatient hospitalization and outpatient care for violent victimization. We replicated the well-established age-crime curve amongst SLCP study members. We found that hospitalization for severe violent victimization was most likely to occur between 20 and 40 years of age. We additionally considered how childhood problem behaviour impacted overall risk and life-course patterning of offending and victimization. Childhood problem behaviour was associated with a greater risk of criminal conviction. But childhood problem behaviour showed inconsistent associations with risk for police suspicion. Childhood problem behaviour was generally associated with greater involvement in crime up to middle adulthood. Childhood problem behaviour was generally associated with a greater risk of victimization. However, we were limited in our ability to estimate the effect of childhood problem behaviour on life-course patterning of victimization due to the rarity of victimization. These results imply a need for larger studies on violent victimization and greater nuance in our understanding of childhood risks and their life-long outcomes.

Background: Violent criminality is at least moderately heritable, but the mechanisms behind this remain largely unexplained. Height, a highly heritable trait, may be involved but no study has estimated the effect of height on crime while simultaneously accounting for important demographic, biological and other heritable confounders.

Methods: We linked nationwide, longitudinal registers for 760 000 men who underwent mandatory military conscription from 1980 through 1992 in Sweden, to assess the association between height and being convicted of a violent crime. We used Cox proportional hazard modelling and controlled for three types of potential confounders: physical characteristics, childhood demographics and general cognitive ability (intelligence).

Results: In unadjusted analyses, height had a moderate negative relationship to violent crime; the shortest of men were twice as likely to be convicted of a violent crime as the tallest. However, when simultaneously controlling for all measured confounders, height was weakly and positively related to violent crime. Intelligence had the individually strongest mitigating effect on the height-crime relationship.

Conclusions: Although shorter stature was associated with increased risk of violent offending, our analyses strongly suggested that this relationship was explained by intelligence and other confounding factors. Hence, it is unlikely that height, a highly heritable physical characteristic, accounts for much of the unexplained heritability of violent criminality.

The Dunedin Multidisciplinary Health and Development Study are an ongoing longitudinal investigation of health and behavior of a complete birth cohort that was drawn from the greater Dunedin metropolitan area, located in the southern coastal region of New Zealand's South Island. This chapter describes the methods used to develop and carry out the Dunedin Study, and how research using the Dunedin Study data is conducted. Location data are used by a "sample tracer" who is employed at each assessment to track, locate, and recruit Study members. The Dunedin Study uses a prospective-longitudinal, correlational design. The basic strategy of the Dunedin Study involves testing for causal relations within this correlational design. The Dunedin Study has comprehensive biomarkers of metabolic, liver, kidney, cardiovascular, dental, immune, and respiratory function, facial aging, and telomere length measured repeatedly at ages 26, 32, 38, and 45 years.

Importance : Health disparities between African Americans and Whites have persisted in the United States. Researchers have recently hypothesized that the relatively poor health of African Americans may be caused, in part, by African American overrepresentation in the criminal justice system.

Objectives: To test the hypothesis that criminal justice system involvement is associated with poor health and greater health risk when controlling for unobserved family factors through a discordant sibling design.

Methods: Subjects were drawn from the Carolina African American Twin Study of Aging (CAATSA). Criminal conviction records were extracted from North Carolina’s Department of Public Safety. Six measures of health and one measure of health risk were analyzed. The health of convicted respondents was compared to that of unrelated non-convicted respondents matched on childhood and demographic factors (“matched sample”). Convicted respondents were also compared to non-convicted siblings (“discordant sibling sample”).

Results: The matched sample included 134 CAATSA respondents. On average, convicted CAATSA respondents, compared to matched non-convicted respondents, were in worse health. Convicted respondents had worse mean self-reported health, worse lung function, more depressive symptoms, and smoked more. The discordant sibling sample included 74 respondents. Convicted siblings and non-convicted siblings had similar self-reported health, depressive symptoms, and smoking. In general, non-convicted siblings were in worse health than non-convicted respondents from the matched sample, implying that poor health runs in families.

Conclusions: This study provided preliminary evidence that some of the association between a criminal record and poor health is confounded by family factors. Though more research is needed to support these results, the study suggests that criminal involvement may not be associated with the surfeit of health problems observed among African Americans. The criminal justice system, nonetheless, could be used to decrease the health disparity.

There is widespread concern in both the popular and academic literature that a surplus of men in a population intensifies mating competition between men, particularly unpartnered men, resulting in increased violence towards both men and women. Recent contributions challenge this perspective and argue that male mating competition and levels of violence will be higher when sex ratios are female-skewed. Existing empirical evidence remains inconclusive. We argue that this empirical ambiguity results from analyses of aggregate-level data, which put inferences at risk of ecological fallacies. Our analysis circumvents such problems by using individual-level, longitudinal demographic register and police data for the Stockholm metropolitan area, Sweden (1990–2003, n = 758,498). These data allow us to investigate the association between municipality-level sex ratios and violent offending (homicide, assault, threat, and sexual crimes) while adjusting for sociodemographic factors. Results suggest that aggregated offending rates are negatively associated with male-skewed sex ratios, whereas individual-level violent offending correlates positively with male-skews. We find that the more-men-more-violence association holds particularly for male violence against other men, but is insignificant for violence against women. Moreover, the association is significant among childless men, but not among fathers. However, robustness checks question the causality of these associations. Female violent offending is positively, albeit due to a low number of cases, insignificantly associated with male-skews. Moreover, both male and female non-violent offending is higher in male-skewed municipalities. We discuss the implications with regard to the theoretical debate and problems of unobserved heterogeneity in the sex ratio literature.

Background: Intellectual disability (ID), schizophrenia spectrum disorder (SSD), bipolar disorder (BD), substance use disorder (SUD), and other mental disorders (OMDs) are associated with increased risks of criminality relative to sex-matched individuals without these conditions (NOIDMD). To resource psychiatric, addiction, and social services so as to provide effective treatments, further information is needed about the size of sub-groups convicted of crimes, recidivism, timing of offending, antecedents, and correlates. Stigma of persons with mental disorders could potentially be dramatically reduced if violence was prevented.

Methods: A birth cohort of 14,605 persons was followed to age 64 using data from Swedish national health, criminal, and social registers.

Results: Percentages of group members convicted of violence differed significantly: males NOIDMD, 7.3%, ID 29.2%, SSD 38.6%, BD 30.7%; SUD 44.0%, and OMD 19.3%; females NOIDMD 0.8%, ID 7.7%, SSD 11.2%, BD 2.4%, SD 17.0%, and OMD 2.1%. Violent recidivism was high. Most violent offenders in the diagnostic groups were also convicted of non-violent crimes. Prior to first diagnosis, convictions (violent or non-violent) had been acquired by over 90% of the male offenders and two-thirds of the female offenders. Physical victimization, adult comorbid SUD, childhood conduct problems, and adolescent substance misuse were each associated with increased risks of offending.

Conclusion: Sub-groups of cohort members with ID or mental disorders were convicted of violent and non-violent crimes to age 64 suggesting the need for treatment of primary disorders and for antisocial/aggressive behavior. Many patients engaging in violence could be identified at first contact with clinical services.

BACKGROUND: Intellectual disability (ID), schizophrenia spectrum disorder (SSD), bipolar disorder (BD), substance use disorder (SUD), and other mental disorders (OMDs) are associated with increased risks of criminality relative to sex-matched individuals without these conditions (NOIDMD). To resource psychiatric, addiction, and social services so as to provide effective treatments, further information is needed about the size of sub-groups convicted of crimes, recidivism, timing of offending, antecedents, and correlates. Stigma of persons with mental disorders could potentially be dramatically reduced if violence was prevented.

METHODS: A birth cohort of 14,605 persons was followed to age 64 using data from Swedish national health, criminal, and social registers.

RESULTS: Percentages of group members convicted of violence differed significantly: males NOIDMD, 7.3%, ID 29.2%, SSD 38.6%, BD 30.7%; SUD 44.0%, and OMD 19.3%; females NOIDMD 0.8%, ID 7.7%, SSD 11.2%, BD 2.4%, SD 17.0%, and OMD 2.1%. Violent recidivism was high. Most violent offenders in the diagnostic groups were also convicted of non-violent crimes. Prior to first diagnosis, convictions (violent or non-violent) had been acquired by over 90% of the male offenders and two-thirds of the female offenders. Physical victimization, adult comorbid SUD, childhood conduct problems, and adolescent substance misuse were each associated with increased risks of offending.

CONCLUSION: Sub-groups of cohort members with ID or mental disorders were convicted of violent and non-violent crimes to age 64 suggesting the need for treatment of primary disorders and for antisocial/aggressive behavior. Many patients engaging in violence could be identified at first contact with clinical services.

In their original article, Burt and Simons (2014) argued that heritability studies should be abandoned because twin and adoption research is a fatally flawed paradigm. They pointed optimistically to epigenetics research as the way forward. In our view, both recommendations are hasty. This commentary will put forward two contrarian opinions. First, twin and adoption studies still have a lot to offer criminologists who seek the social causes of crime. Second, epigenetics research has very little to offer yet for criminologists who seek the social causes of crime.

Research on maturation and its relation to antisocial behavior has progressed appreciably in recent years. Psychosocial maturation is a relatively recent concept of development that scholarship has linked to risky behavior. Psychosocial maturation appears to be a promising explanation of the process of exiting criminal behavior, known as desistance from crime. However, to date, research has not examined whether psychosocial maturation is related to desistance in similar ways across race/ethnicity. Using the Pathways to Desistance Study which followed a mixed-race/ethnicity group of serious adolescent offenders for 7 years, this research tested growth in psychosocial maturation across race/ethnic groups. The sample (14.46% female, average age 15.97 at baseline) was composed of white (n = 250), black (n = 463), and Hispanic (n = 414) individuals. The results showed variation in trajectories of psychosocial maturation with blacks having higher initial levels but slower growth in maturation over time compared to whites. Psychosocial maturation was negatively related to crime across all racial/ethnic groups. Across all racial/ethnic groups, differences in the magnitude of the association between psychosocial maturation and desistance were small. Rather than needing distinct theories for specific groups, psychosocial maturation appears to be a general theoretical perspective for understanding desistance from crime across races/ethnicities. Policy formulation based on psychosocial maturation would, therefore, be applicable across racial/ethnic groups.

Background: Youth offenders have a high risk of being injured or dying prematurely. However, few studies have considered the role of imprisonment and potential childhood risk factors for these high rates.

Aim: To examine the risk of unintentional injury and premature death in non-imprisoned and imprisoned youth offenders, and to examine the role of parental criminal convictions and psychiatric disorders and own childhood psychiatric disorders.

Methods: All individuals (N = 1,839,711) born in Sweden between 1978 and 1996 were identified using Swedish population-based registers. The exposure was criminal conviction between ages 15-20 years of age.

Results: Imprisoned youth offenders had the highest risk for unintentional injury (HR = 2.29 [2.19-2.40]) and premature death (HR = 10.76 [9.52-12.16]), followed by nonimprisoned youth offenders, compared to non -convicted youth. All childhood risk factors increased the risk for these outcomes among non-imprisoned youth offenders. Among imprisoned youth offenders, parental criminal convictions and parental psychiatric disorders increased the risk for unintentional injury, and parental psychiatric disorders and own childhood psychiatric disorders increased the risk for premature death.

Conclusions: Our study shows there are robust modifiable childhood risk factors for injury and mortality among youth offenders. However, the importance of them to assess risk may differ between non-imprisoned and imprisoned youth offenders.

Background: Psychiatric diagnoses are important risk factors for criminal convictions, but few longitudinal studies have examined comorbidity patterns in relation to youth criminal convictions.

Aim: To explore associations between specific psychiatric diagnoses (substance use disorder (SUD), ADHD, depression, PTSD, intellectual disabilities (ID), and autism spectrum disorders (ASD)) and comorbidities of internalizing, externalizing, or neurodevelopmental diagnoses (NDD) in relation to risk of non-violent or violent criminal convictions in youth, including potential sex differences.

Methods: Data on 1,411,538 individuals born in Sweden (1985–1998) were obtained from national population-based registers. Exposure was psychiatric diagnoses and outcome was criminal convictions between ages 15 and 20.

Results: 17% of individuals had a psychiatric diagnosis, of whom 20% were convicted of a crime. All diagnoses, except ID and ASD, increased the risk of non-violent and violent crimes. Comorbidities of externalizing and internalizing diagnoses heightened the risk compared to single diagnoses. NDD increased the risk among SUD, depression, and PTSD, while NDD comorbid with another NDD decreased the risk for criminal convictions.

Conclusion: Of the three comorbidity categories, externalizing disorders heightened risk the most, followed by internalizing disorders. This study highlights specific risk patterns for criminal convictions related to comorbidities, and to crime type and sex.

Drawing on psychological and sociological theories of crime causation, we tested the hypothesis that genetic risk for low educational attainment (assessed via a genome-wide polygenic score) is associated with criminal offending. We further tested hypotheses of how polygenic risk relates to the development of antisocial behavior from childhood through adulthood. Across the Dunedin and Environmental Risk (E-Risk) birth cohorts of individuals growing up 20 years and 20,000 kilometers apart, education polygenic scores predicted risk of a criminal record with modest effects. Polygenic risk manifested during primary schooling in lower cognitive abilities, lower self-control, academic difficulties, and truancy, and it was associated with a life-course-persistent pattern of antisocial behavior that onsets in childhood and persists into adulthood. Crime is central in the nature-nurture debate, and findings reported here demonstrate how molecular-genetic discoveries can be incorporated into established theories of antisocial behavior. They also suggest that improving school experiences might prevent genetic influences on crime from unfolding.